Correction: TNF/TNFR signal transduction pathway-mediated anti-apoptosis and anti-inflammatory effects of sodium ferulate on IL-1β- induced rat osteoarthritis chondrocytes in vitro

نویسندگان

  • Jun Qin
  • Liang Shang
  • An-song Ping
  • Jing Li
  • Xiao-jun Li
  • Hong Yu
  • Jacques Magdalou
  • Liao-bin Chen
  • Hui Wang
چکیده

After publication of our recent article [1], it was brought to our attention that an inaccurate observation was made in the Results section. We stated that diff erent concentrations of SF have no eff ect on normal chondrocytes viability. However, Figure 1 demonstrates that SF concentra tions 500 and 1000 μmol/L signifi cantly increase chondrocytes viability compared with the control group. Th e following sentence has now been deleted from the Results section: “All of these results show that diff erent concentrations of SF from 125 to 1,000 μmol/l have no eff ect on normal chondrocyte viability”.

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

منابع مشابه

Response to 'TNF/TNFR signal transduction pathway-mediated anti-apoptosis and anti-inflammatory effects of sodium ferulate on IL-1β-induced rat osteoarthritis chondrocytes in vitro' - authors' reply

Arthritis Research and Th erapy could draw such great interest from the readers. Th e following is our response to Ma and colleagues' concerns [1]. In response to the fi rst question, at the beginning of the research we had screened the entire apoptotic pathways using the Rat Apoptosis RT2 Profi ler™ PCR Array (SABiosciences, Frederick, MD, USA), and the result (Table 1 in the original paper [2...

متن کامل

TNF/TNFR signal transduction pathway-mediated anti-apoptosis and anti-inflammatory effects of sodium ferulate on IL-1β-induced rat osteoarthritis chondrocytes in vitro

INTRODUCTION Sodium ferulate (SF) is a natural component of traditional Chinese herbs. Our previous study shows that SF has a protective effect on osteoarthritis (OA). The objective of this study was to investigate the effect of SF on the TNF/TNF receptor (TNFR) signal transduction pathway of rat OA chondrocytes. METHODS Primary rat articular chondrocytes were co-treated with IL-1β and SF. Ch...

متن کامل

Response to 'TNF/TNFR signal transduction pathway-mediated anti-apoptosis and anti-inflammatory effects of sodium ferulate on IL-1β-induced rat osteoarthritis chondrocytes in vitro'

and colleagues, in which they addressed, on the basis of their previous work, the underlying mechanisms for the protective eff ect of a small component of traditional Chinese herbs, sodium ferulate (SF), on osteoarthritis (OA) [1]. Utilizing the classical in vitro OA chondrocyte model induced by IL-1β, they provided lines of evidence that SF does make a big diff erence to the OA model in a dose...

متن کامل

Asiaticoside attenuates hyperoxia-induced lung injury in vitro andin vivo

Objective(s): Asiaticoside (AS) displays anti-inflammation, and anti-apoptosis effect, but the role of AS in hyperoxia-induced lung injury (HILI) treatment is undefined. Therefore, the aim of this study was to investigate the effects of AS on HILI on premature rats and alveolar type II (AEC II) cells.Materials and Methods: Sprague-Dawley...

متن کامل

The anti-inflammatory effects of venlafaxine in the rat model of carrageenan-induced paw edema

Objective(s):Recently anti-inflammatory effects of antidepressants have been demonstrated. Venlafaxine belongs to newer antidepressants with serotonin norepinephrine reuptake inhibition property. The pain alleviating properties of venlafaxine in different pain models such as neurogenic pain, diabetic neuropathy, and fibromyalgia have been demonstrated. Anti-inflammatory effects of venlafaxine a...

متن کامل

ذخیره در منابع من


  با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید

برای دانلود متن کامل این مقاله و بیش از 32 میلیون مقاله دیگر ابتدا ثبت نام کنید

ثبت نام

اگر عضو سایت هستید لطفا وارد حساب کاربری خود شوید

عنوان ژورنال:

دوره 15  شماره 

صفحات  -

تاریخ انتشار 2013